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To discern whether the lipolytic effects of AOD 9604 are solely contingent upon enhanced β3-AR expression, further investigations were conducted utilizing mice with genetically ablated lipolytic receptors

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Human frame shift mutations affecting the carboxyl terminus of perilipin increase lipolysis by failing to sequester the adipose triglyceride lipase (ATGL) coactivator AB-hydrolase-containing 5 (ABHD5)